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Palabras clave – provistas por la editorial

n/a; analgesia; quantitative phosphoproteomics; neurodegeneration; neuron; neuronal circuits; mating; sex-specific differences; AMPA receptor complex; parallel reaction monitoring; splicing; phosphorylation; peptidylglycine ?-amidating monooxygenase; pherophorin; Alzheimer’s disease; neuroscience; yohimbine; synapses; adolescence; conformational antibody; opioid receptors; protein interaction networks; Alzheimer’s Disease; transmembrane AMPA receptor regulatory protein; neuroproteome; ethanol; U1 snRNP; mass spectroscopy; morphine; protein phosphatase-1; neuroproteomics; axons; reinstatement; DIA; voltage gated channels; prohormone convertase; postsynaptic density; neuroimmune; dendrites; carboxypeptidase; GPCR signaling; CaMKII; FGF14; SynCAM; nicotinic receptor; synaptic plasticity; plasma; serum; corticosterone; dopamine; Receptor-type tyrosine-protein phosphatase zeta; Ptprz1; PRM; cell type; schizophrenia; laser capture microdissection; synapse specificity; LC-MS/MS; mouse; drug abuse; synaptic cleft; striatum; Cadm; cytokine; spinophilin; PSD; trans-synaptic adhesion; nicotine; proximity labeling; biomarkers; addiction; PKA; synapse; proteome; signal peptide; ventral tegmental area; protein aggregation; cerebrospinal fluid; bioinformatics; progressive ratio; interactome; subtilisin; affinity chromatography; basal ganglia; cocaine; targeted proteomics; postsynaptic; mass spectrometry; connectome; quantitative mass spectrometry; biotinylation; proteomics; amphetamine; autism; data-independent acquisition; excitatory/inhibitory tone; neuropeptidomics; matrix metalloproteinase; cilia; R-PTP-zeta; cognitive impairment